Calcium channel blockers

Which of the following calcium channel blockers is specifically identified for use in preventing neurologic damage due to cerebral vasospasm?

What is the primary mechanism of action for calcium channel blockers?

Which two calcium channel blockers are specifically mentioned to decrease AV nodal conduction in addition to their primary actions?

Most calcium channel blockers are contraindicated in decompensated heart failure, except for which two agents?

Concurrent use of calcium channel blockers with beta blockers, digoxin, clonidine, and ivabradine may lead to what additive effect?

Which two drugs can increase serum digoxin levels and potentially cause toxicity when used concurrently?

What is a key patient instruction regarding the administration of sustained-release calcium channel blocker capsules?

What important patient teaching point is related to dental hygiene for those taking calcium channel blockers?

When assessing a patient on long-term verapamil or diltiazem therapy for arrhythmias, what should be monitored continuously during IV therapy and periodically thereafter?

What is a major contraindication for the use of most calcium channel blockers, related to heart rhythm and function?

What advice should be given to a patient taking calcium channel blockers to minimize orthostatic hypotension?

For a patient receiving IV verapamil or diltiazem for arrhythmias, what is a crucial nursing assessment?

The effectiveness of calcium channel blockers may be increased by concurrent use with which of the following medications?

What is one of the desired outcomes for a patient being treated with a calcium channel blocker for angina?

Which of the following is NOT listed as a calcium channel blocker used in the treatment of hypertension?

Which factor may enhance the hypotensive effects of calcium channel blockers, requiring a patient to be cautious?

Which potential nursing diagnosis is listed as being related to the indications for calcium channel blocker therapy?

What is the primary effect of nimodipine on blood vessels?

Under the 'Precautions' section, in which patient population should calcium channel blockers be used cautiously?

A nurse is assessing a patient taking a calcium channel blocker. Which of the following signs would suggest the development of heart failure (HF)?

Besides 2nd- or 3rd-degree heart block, what other condition is listed as a contraindication for calcium channel blockers like verapamil and diltiazem?

What should a patient taking calcium channel blockers be advised to do before taking any over-the-counter (OTC) medications?

Which of these is NOT an expected therapeutic use of verapamil?

How do phenytoin and phenobarbital interact with calcium channel blockers?

What assessment should a nurse perform for a patient with angina who is taking a calcium channel blocker?

According to the text, what is a primary general action of calcium channel blockers on coronary arteries?

What general instruction about medication adherence should be given to patients taking calcium channel blockers?

For which reason would a patient taking a calcium channel blocker be advised to carry identification describing their medication regimen?

What is the desired outcome related to neurological deficits for a patient treated with a calcium channel blocker for vasospasm?

Which statement about the administration of calcium channel blockers with regard to meals is correct?

What is a key component of the nursing assessment for a patient on nimodipine for cerebral vasospasm?

Among the calcium channel blockers listed, which one is NOT indicated for the treatment and prophylaxis of angina pectoris or coronary artery spasm?

A patient asks why they need to inform their surgeon about taking a calcium channel blocker before a procedure. What is the most appropriate reason?

Which of the following is NOT a sign of heart failure (HF) that a nurse should routinely assess for in a patient taking a calcium channel blocker?

A patient is started on a calcium channel blocker for hypertension. Which outcome indicates the therapy is effective?

What is the safety status of calcium channel blockers during pregnancy and lactation according to the text?

When is ECG monitoring for arrhythmias specifically recommended for patients on long-term therapy with calcium channel blockers?

What is the potential interaction between verapamil/diltiazem and digoxin?

Which condition is NOT listed as a general use for calcium channel blockers in the provided text?

A patient taking a calcium channel blocker for hypertension should be encouraged to comply with which additional intervention?

What is the primary reason for monitoring intake and output ratios and daily weight in a patient taking a calcium channel blocker?

What is a key desired outcome for a patient taking a calcium channel blocker for supraventricular tachyarrhythmias?

Among the following, which agent is NOT listed as causing additive bradycardia when used with calcium channel blockers?

Which patient populations should receive calcium channel blockers with caution, as stated in the 'Precautions' section?

A patient with angina is also prescribed concurrent nitrate therapy. What instruction is important for this patient?

What potential side effect of calcium channel blockers necessitates the patient teaching point about seeing a dentist frequently for cleanings?

What is the consequence of opening, crushing, or chewing sustained-release calcium channel blocker capsules?

A patient with a history of decompensated heart failure is prescribed a calcium channel blocker. Which of the following would be an appropriate choice according to the text?

What is the rationale for assessing a patient's neurological status when they are taking nimodipine?

An increased risk of digoxin toxicity is associated with the concurrent use of digoxin and which two calcium channel blockers?