What is the reason given for stunted fetal growth and maturation in cases of uncontrolled maternal type 1 diabetes?
Explanation
This question probes the underlying cause for poor fetal outcomes in uncontrolled maternal type 1 diabetes, linking it to the mother's overall metabolic health.
Other questions
What is the primary source of energy for the fetus before birth?
For how long is the glucose stored as glycogen in a newborn's body sufficient to supply its needs after birth?
What is the primary reason for the prevention of significant gluconeogenesis in the liver of a neonate?
What often happens to an infant's blood glucose concentration on the first day after birth due to limited gluconeogenesis?
To what level can a neonate's blood glucose concentration frequently fall on the first day of life?
Why is the glucose concentration in the neonate's blood described as unstable and low during the first week of life?
What condition develops in the islets of Langerhans of an infant born to an untreated diabetic mother?
What is the immediate consequence for the infant's blood glucose shortly after birth due to hypertrophy and hyperfunction of the islets of Langerhans?
In an infant born to an untreated diabetic mother, to what level might the blood glucose concentration fall shortly after birth?
What is a notable finding about the clinical effects of very low blood glucose concentration in a neonate compared to an adult?
What does the text identify as the most common cause of large babies (macrosomia)?
How is maternal type 2 diabetes characterized in the text?
In a mother with type 2 diabetes, what is believed to stimulate fetal growth and contribute to increased birth weight?
What two factors may contribute to increased fetal growth in the case of a mother with type 2 diabetes?
In a large baby born to a mother with type 2 diabetes, what is the primary composition of the increased fetal weight?
In infants of mothers with type 2 diabetes who experience increased fetal weight, what is the typical effect on body length?
What term is used in the text to describe the increased size of some organs that may occur in the fetus of a mother with type 2 diabetes?
What is the potential effect on fetal growth when a mother has uncontrolled type 1 diabetes?
According to the text, what causes uncontrolled type 1 diabetes?
What effect does consuming a high-carbohydrate diet have on muscle glycogen stores compared to a mixed diet?
According to the data provided, what are the approximate muscle glycogen stores for a person on a high-fat diet?
What are the approximate muscle glycogen stores for a person who consumes a high-carbohydrate diet, according to the provided data?
Based on the data provided, muscle glycogen stores on a high-carbohydrate diet are approximately how many times greater than on a high-fat diet?
How does diet composition affect endurance, as measured by the time an athlete can sustain a race until exhaustion?
What is the approximate endurance time, until complete exhaustion, for an athlete on a mixed diet?
What is the approximate endurance time, until complete exhaustion, for an athlete who has consumed a high-carbohydrate diet?
During the first few seconds or minutes of exhaustive exercise, from where is most of the energy derived?
In endurance athletic events lasting longer than 4 to 5 hours, what does the muscle primarily depend on for energy after its glycogen stores are depleted?
At the time of exhaustion during a prolonged exercise event, what percentage of energy is being derived from fats rather than carbohydrates?
Besides stored muscle glycogen, what is another major source of carbohydrate energy for muscles during exercise?
During prolonged athletic events like marathons, what percentage of the required energy can be provided by glucose solutions given to an athlete to drink?
What is the primary dietary message for athletes before participating in a grueling athletic event?
Following exhaustive exercise, approximately how long does it take for full recovery of muscle glycogen for a person on a high-carbohydrate diet?
Under which condition does the text show very little recovery of muscle glycogen even after 5 days?
After intense exercise, what happens to the remaining lactic acid in the body once energy is available from oxidative metabolism?
How does the rate of ATP formation from the glycogen-lactic acid system (anaerobic glycolysis) compare to the rate from the oxidative mechanism of mitochondria (aerobic system)?
Which energy system is used by muscles for maximal power surges lasting a few seconds, such as in a 100-meter dash or weight lifting?
For which duration of maximal muscle activity is the glycogen-lactic acid system primarily used?
What is the effect of maintaining body fitness through exercise on the risk of developing chronic metabolic disorders like type 2 diabetes?
Besides type 2 diabetes, what other chronic metabolic disorder associated with obesity does the text mention is reduced by exercise and body fitness?
What is the specific effect of moderate exercise on the body's response to insulin?
Under what key condition has moderate exercise been shown to improve insulin sensitivity?
What is a potential clinical impact of improved insulin sensitivity from moderate exercise for patients with type 2 diabetes?
What does the text conclude about the relationship between the beneficial effects of regular exercise on chronic diseases and changes in body weight or adiposity?
Which of the following statements about exercise and type 2 diabetes is best supported by the provided text?
According to the text, what is the most important factor that determines muscle endurance during prolonged exercise?
What is the key recommendation given to athletes regarding their activity level in the 48 hours preceding a grueling event?
How does the text describe the difference in clinical outcomes between a neonate and an adult experiencing very low blood glucose levels (e.g., lower than 20 mg/dl)?
Based on the data for diet and endurance provided in Chapter 85, which of the following conclusions is accurate?