What is the primary effect of high concentrations of glucagon on gastric secretion?
Explanation
This question focuses on the specific inhibitory role of high levels of glucagon on the secretory function of the stomach.
Other questions
Which cells are the main source of the hormone Ghrelin, which is known to stimulate appetite?
Under what physiological condition do the blood levels of Ghrelin typically rise, suggesting its role in stimulating feeding?
What type of nutrients entering the duodenum act as the primary stimulus for the release of Cholecystokinin (CCK)?
CCK contributes to satiation and meal cessation by activating receptors on local sensory nerves in the duodenum, which then send messages to the brain via which specific nerve?
What is the main function of Cholecystokinin (CCK) regarding the regulation of eating behavior during a meal?
From which parts of the gastrointestinal tract is the hormone Peptide YY (PYY) primarily secreted?
How long after a meal is ingested do the blood concentrations of Peptide YY (PYY) typically take to reach their peak levels?
Meals with a high content of which specific macronutrient are observed to cause higher peak levels of Peptide YY (PYY)?
The hormones GLP-1 and GIP are often referred to by what collective name because they enhance insulin release in response to plasma glucose?
In addition to enhancing insulin release, what effect do the incretins GLP-1 and GIP have on glucagon secretion from the alpha cells of the pancreas?
What is the purpose of the 'anticipatory' increase in blood insulin caused by gastrointestinal hormones like incretins after a meal?
What effect does the hormone aldosterone have on the composition of secretions from salivary glands?
In which specific part of the intestine does aldosterone most significantly enhance sodium absorption?
According to the text, what is a potential consequence related to the alimentary tract in a state of aldosterone deficiency?
High concentrations of the hormone glucagon can inhibit the secretion of which substance from the stomach?
In addition to its effects on glucose metabolism, high concentrations of glucagon are mentioned to enhance which secretion related to the liver and gallbladder?
What is the approximate amount of calcium that is estimated to enter the intestines each day through secreted gastrointestinal juices and sloughed mucosal cells?
Which of the following gastrointestinal hormones is specifically identified as being released mainly from the duodenum?
Which of the following is NOT a gastrointestinal function influenced by Cholecystokinin (CCK)?
Salivation is mentioned as a mechanical process of feeding controlled by centers in what part of the nervous system?
What is the relationship between food intake and the secretion of gastrointestinal hormones like PYY and CCK?
Which hormone has an effect on salivary and intestinal epithelial cells that is similar to its effect on the renal tubules, leading to conservation of body salt?
What is the primary function of the effect of incretins like GLP-1 and GIP on insulin secretion?
Which two gastrointestinal hormones are specifically mentioned as having opposing effects on appetite?
What is the effect on the rate of insulin secretion when excess amino acids are administered at the same time as blood glucose is elevated?
According to the text, what happens to the intestinal absorption of phosphate?
How does chronic administration of CCK affect body weight, according to the text?
Besides the ileum and colon, where else is Peptide YY (PYY) secreted from?
In the experimental context of PYY injections into mice, what was the duration of the observed decrease in food intake?
The text states that glucagon-like peptide (GLP) and insulin both tend to have what effect on appetite?
What is the primary role of aldosterone's effect on the salivary glands?
Which of the following is listed as a gastrointestinal hormone that can cause a moderate increase in insulin secretion?
Signals from the gastrointestinal tract that provide sensory information about stomach filling are received by which part of the brain to help regulate food intake?
Which hormone is specifically mentioned as being part of a feedback mechanism for controlling food intake in the provided Figure 72-1, but is secreted by the pancreas, not the gut wall?
The action of incretins like GLP-1 and GIP can almost double the rate of insulin secretion, but this potentiating effect is dependent on what other condition?
What happens to Ghrelin levels in the blood just before a meal?
Which gastrointestinal hormone that suppresses feeding is secreted from the entire GI tract but most notably from the ileum and colon?
What is the primary stimulus for the intestinal secretion of glucagon-like peptide (GLP)?
The hormone aldosterone can prevent the loss of sodium in the stools by enhancing its absorption in which part of the alimentary tract?
In addition to suppressing appetite and enhancing insulin release, what is the third action of incretins GLP-1 and GIP on the islets of Langerhans?
Which of the following gastrointestinal hormones is noted to have a short-lived effect and functions mainly to prevent overeating during a single meal?
The release of which gastrointestinal hormone is stimulated by fasting and inhibited by food ingestion?
The action of aldosterone on intestinal epithelial cells is crucial for preventing the loss of which substance in the feces?
Which of the following is an accurate description of the effect of the incretins GLP-1 and GIP?
What is the status of the scientific understanding regarding the importance of Peptide YY (PYY) in regulating appetite in humans?
Besides the oxyntic cells of the stomach, where else is the hormone Ghrelin secreted, although to a much lesser degree?
Which of these gastrointestinal hormones that suppresses feeding is released mainly from the duodenum?
The failure of the intestines to properly absorb sodium chloride and water, as seen in aldosterone deficiency, can lead to what condition?
High concentrations of glucagon are noted to have what two effects on alimentary tract secretions?